Important solved Questions for NBDE part2 -3

QUESTION: Hairy tongue – hypertrophy filly form papilla.
Ans. Hairy tongue is a beningn condition of the tongue.
Poor oral hygiene
Extended use of antibiotics
Corticosteroids
Hydrogen peroxide
Smoking
Hairy leukoplakia is a non malignant lesion seen exclusively in AIDS PATIENT
20) which of the following is seen with hyperplastic foliate papillae?
Ans. Lingual tonsil hyperplasia

QUESTION: Loss of filliform papilla- vitamin def- vit b

QUESTION: Bilateral swelling of parotid cannot be caused by: Anorexia
Metabolic conditions associated with bilateral parotid gland enlargement include diabetis
mellitus,hypertension,sjogrens syndrome,alcoholism.

QUESTION: Patient has bilateral white lines @ occlusal plane, what is primary microscopic
finding?
Epithelial hyperkeratosis

QUESTION: Pt has hyperkeratosis around occlusal? linea albaQUESTION: What is white and bilateral on buccal mucosa (leukoedema not choice), Linea
Alba

QUESTION: Ulcer on tongue repeated every 4 months- apthous ulcer

QUESTION: why brush tongue – to reduce odor

QUESTION: Pic: had a red thing on tongue where is it from (candidiasis, Kaposi, syphilis,
gonnaria) candidiasis

QUESTION: Behçet’s disease Pic of something on tongue: aphthous ulcer –
Apthous ulcer – common in behcets syndrome & apthous ulcer

BEHCETS SYNDROME- It is a rare disorder that causes chronic inflammation in bloos vessels
throughout the body
Oral & genital apthous ulcers
Conjunctivitis, uveitis,arthritis, headache complication blindness

QUESTION: Pathognomonic for measles? Koplik’s spots………buccual mucosa ulcerated

QUESTION: Transillumination in children – Koplik? (Koplik’s spots are associated with
measles)

QUESTION: Syphilis: hutchinson triad (presentation for congenital syphilis, and consists of
three
phenomena: interstitial keratitis, Hutchinson incisors, and eighth nerve deafness.

QUESTION: indents on incisal edge with narrowing at mesial and distal? I guessed congenital
syphilis (Hutchinson’s tooth?) screw driver incisors

QUESTION: A chanker due to Syphilis mostly resembles: herpes

QUESTION: stages of syphilis is most infectious: primary, secondary, tertiary,
Secondary stage is most infectious with mucopapular rash
Condylomata lata is seen in secondary stage

QUESTION: Heck disease: 13 and 32
(also known as focal epithelial hyperplasia) multiple dome shaped warts on oral mucosa
human papilloma virus types 13 and 32.

QUESTION: baby with streaks on palate• bone nodulus
• Epstein pearls
• congenital epulus

QUESTION: neonate with a bunch of nodules on alveolar ridge. What is it?
a. Bohn’s nodule
– keratin-filled cysts of salivary gland origin on palate of newborn

Oral Pathology:
Lupus Erythematosus:
Chronic autoimmune disease
Discoid lupus erythematosus…. chronic skin conditions of sores & inflammation scarring of
face ,ears,scalp & oral mucosa

ORAL LESIONS MIMIC EROSIVE LICHEN PLANUS
Systemic lupus erythematosus…chronic connective tissue disorderthat involves oragans
including kidneys,heart,joints,skin,mucous membrane,&blood vessels
BUTTERFLY SHAPED RASH

QUESTION: Xerostomia, complication of :Sjögren’s syndrome, dry moth dry eye ,enlargement
of parotid &submandibular bilatareally
Cavernous Sinus Thrombosis:

QUESTION: cavernous sinus problem – due to infection of upper lip / canine space infxn / max
ant
Teeth

QUESTION: Most likely to cause cavernous sinus thrombosis: valve infected by endocarditis,
soft tissue abcess of upper lip
Nerves invoved are OCCULOMOTOR, TROCHLER ,ABDUCENS,TRIGEMINAL ,
(OPTHALMAIC,&MAXILLARY

QUESTION: Site of infection most likely to enter cavernous sinus? Anterior triangle

CystQUESTION: Danger triangle of the face – cavernous sinus (no valves in the veins)

QUESTION: Why are you afraid of having infection in anterior triangle (i.e. upper lip) because
there
are valve-less veins that can send infection back to the brain

QUESTION: Which of the following causes Cavernous sinus thrombosis: A)Subcutaneous
Abscess of
upper lip
Infections in upper front teeth are within the area of the face known as the “dangerous triangle”.
The dangerous triangle is visualized by imagining a triangle with the top point about at the bridge of the nose and the two lower points on either corner of the mouth

QUESTION: Danger zone of Cavernous Sinus: Signs and symptoms. What is the first one?
HEAD ACHE( GOOGLE)

QUESTION: first sign of cavernous sinus:
bulging eye??
loss vision
***HEADACHE***

QUESTION: Pathognomonic sign of CST? Ptosis, bulging eye, periorbital edema

QUESTION: Cavernous sinus thrombosis early indication? Peri-orbital swelling, blurry vision

QUESTION: Cavernus sinus has : ptosis, decreased vision, opthalplagia
Cavernous sinus thrombosis (CST) is the formation of a blood clot within the CS at the base of
the brain which drains deoxygenated blood from the brain back to the heart. usually from a
infection from nose, sinuses, ears, teeth or Forunculo. Staphylococcus aureus and Streptococcus
are often the associated. symptoms include: decrease or loss of vision, chemosis, exophthalmos
(bulging), ptosis, headaches(1st one) and paralysis of the cranial nerves which course through the
cavernous sinus. This infection is life-threatening and requires immediate TX.
Ludwigs Angina:

QUESTION: What space is not associated with ludwigs angina? Associated with sublingual,
submental, submandibular

QUESTION: Ludwig’s angina seen in all spaces except: Retropharyngeal

QUESTION: Cellulitis most of the time involves unilateral, ludwigs angina is bilateral and
complication is edema of GLOTTI

QUESTION Bilateral submandibular infection, tongue was elevated due to infection – Ludwigs
angina

QUESTION: What u need to worry most abt ludwigs? swelling of glottis

QUESTION: complication of lugwig’s angina:edema of glottis

QUESTION: Ludwig’s Angina symptoms? Swelling, pain and raising of the tongue, swelling of the neck and the tissues of the submandibular and sublingual spaces, malaise, fever, dysphagia (difficulty swallowing) and, in severe cases, stridor or difficulty breathing.

QUESTION: What is the main danger in Ludwig’s angina? closing of the airway

QUESTION: Mandibular 2nd molar infection spreads to what space? Submandibular space.

QUESTION: Infxn of mnd 2nd pm goes into submandibular space

QUESTION: Premolars and molars infection –submandibular space

QUESTION: Which muscle separates 2 potential infection spaces from a maxillary 2nd molar? Buccinator or Masseter

QUESTION: if you have an infection in the lateral pharyngeal space what muscle is involved?
Medial
Pterygoid
The lateral aspect is more involved, and is bordered by the ramus of the mandible, the deep lobe of
the parotid gland, the medial pterygoid muscle, and below the level of the mandible, the lateral
aspect is bordered by the fascia of the posterior belly of digastric muscle.

QUESTION: You are extracting a mandibular 3rd molar and the distal root disappears into which
68
space? submandibular space

QUESTION: IAN tract infection, ‘-[involves what space? Pterymandibular space
Treacher Collins Syndrome:
Downw=ard facing eyes

QUESTION: Which disorder least developmental delay-trecher collins syndrome

QUESTION: Treacher Collins-loss (hypoplasia) of zygomatic bone, what do patients with
cleidocranialdysplasia have? Loss of clavicle

QUESTION: Malformed ear, mandibular hypoplasia – Treacher Collins

QUESTION: Describes patient saying they have mandibular hypoplasia, Malformed ear
eyelids, ear pinna– Treacher Collins
Scarlet Fever:

QUESTION: Strawberry tongue seen in scarlet fever, Also in Kawasaki disease and toxic shock syndrome
Fordyce Granules:

QUESTION: Fordyce granules – ectopic sebaceous gland

Turner Toot

QUESTION: turners tooth – single tooth affected

QUESTION: Turner’s tooth is caused by: I put “trauma or local infection”

QUESTION: What gives you Turners incisors
• syphilis
• trauma during delivery
• *trauma during pregnancy
TURNERS HYPOPLASIA IS WITH HISTORY OF TRAUMA/INFECTION IN THEIR
PRIMARY PRECEDECESSOR

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